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MA49604B1 - Human antibodies binding to pd-1 - Google Patents

Human antibodies binding to pd-1

Info

Publication number
MA49604B1
MA49604B1 MA49604A MA49604A MA49604B1 MA 49604 B1 MA49604 B1 MA 49604B1 MA 49604 A MA49604 A MA 49604A MA 49604 A MA49604 A MA 49604A MA 49604 B1 MA49604 B1 MA 49604B1
Authority
MA
Morocco
Prior art keywords
antibodies
cell
human antibodies
binds
bind
Prior art date
Application number
MA49604A
Other languages
French (fr)
Other versions
MA49604A1 (en
Inventor
Andrew Murphy
Gavin Thurston
Nicholas Papadopoulos
Ella Ioffe
Elena Burova
Original Assignee
Regeneron Pharma
Priority date (The priority date is an assumption and is not a legal conclusion. Google has not performed a legal analysis and makes no representation as to the accuracy of the date listed.)
Filing date
Publication date
Application filed by Regeneron Pharma filed Critical Regeneron Pharma
Priority claimed from PCT/US2015/012589 external-priority patent/WO2015112800A1/en
Publication of MA49604A1 publication Critical patent/MA49604A1/en
Publication of MA49604B1 publication Critical patent/MA49604B1/en

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  • Medicines That Contain Protein Lipid Enzymes And Other Medicines (AREA)
  • Pharmaceuticals Containing Other Organic And Inorganic Compounds (AREA)

Abstract

La présente invention concerne des anticorps qui se lient à la protéine de mort cellulaire programmée-1 (pd-1) du co-inhibiteur de lymphocyte t, et des méthodes d'utilisation. Selon certains modes de réalisation de l'invention, les anticorps sont des anticorps complètement humains qui se lient à pd-1. Dans certains modes de réalisation, la présente invention concerne des molécules de liaison antigénique multi-spécifiques comprenant une première spécificité de liaison qui se lie à pd-1 et une seconde spécificité de liaison qui se lie à un antigène tissulaire auto-immun, un autre co-inhibiteur de lymphocyte t, un récepteur de fc ou un récepteur de lymphocyte t. Dans certains modes de réalisation, les anticorps de l'invention sont utilisés pour inhiber ou neutraliser l'activité de pd-1, ce qui fournit un moyen de traiter une maladie ou une affection comme le cancer ou une infection virale chronique. Dans d'autres modes de réalisation, les anticorps sont utilisés pour renforcer ou stimuler l'activité de pd-1, ce qui fournit un moyen de traiter, par exemple, une maladie ou une affection auto-immune.The present invention relates to antibodies that bind to t cell co-inhibitor programmed cell death protein-1 (pd-1), and methods of use. According to certain embodiments of the invention, the antibodies are fully human antibodies that bind to pd-1. In some embodiments, the present invention relates to multi-specific antigen binding molecules comprising a first binding specificity that binds to pd-1 and a second binding specificity that binds to an autoimmune tissue antigen, another t-cell co-inhibitor, a fc receptor or a t-cell receptor. In certain embodiments, the antibodies of the invention are used to inhibit or neutralize the activity of pd-1, which provides a means of treating a disease or condition such as cancer or a chronic viral infection. In other embodiments, the antibodies are used to enhance or stimulate pd-1 activity, thereby providing a means of treating, for example, an autoimmune disease or condition.

MA49604A 2014-01-23 2015-01-23 Human antibodies binding to pd-1 MA49604B1 (en)

Applications Claiming Priority (2)

Application Number Priority Date Filing Date Title
US201461930576P 2014-01-23 2014-01-23
PCT/US2015/012589 WO2015112800A1 (en) 2014-01-23 2015-01-23 Human antibodies to pd-1

Publications (2)

Publication Number Publication Date
MA49604A1 MA49604A1 (en) 2022-02-28
MA49604B1 true MA49604B1 (en) 2022-09-30

Family

ID=58698425

Family Applications (1)

Application Number Title Priority Date Filing Date
MA49604A MA49604B1 (en) 2014-01-23 2015-01-23 Human antibodies binding to pd-1

Country Status (2)

Country Link
AR (1) AR099148A1 (en)
MA (1) MA49604B1 (en)

Also Published As

Publication number Publication date
MA49604A1 (en) 2022-02-28
AR099148A1 (en) 2016-07-06

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