AU2001267553A1 - Combined therapy against tumors comprising substituted acryloyl distamycin derivatives, taxanes and/or antimetabolites - Google Patents
Combined therapy against tumors comprising substituted acryloyl distamycin derivatives, taxanes and/or antimetabolitesInfo
- Publication number
- AU2001267553A1 AU2001267553A1 AU2001267553A AU2001267553A AU2001267553A1 AU 2001267553 A1 AU2001267553 A1 AU 2001267553A1 AU 2001267553 A AU2001267553 A AU 2001267553A AU 2001267553 A AU2001267553 A AU 2001267553A AU 2001267553 A1 AU2001267553 A1 AU 2001267553A1
- Authority
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- Prior art keywords
- amino
- methyl
- carbonyl
- pyrrol
- formula
- Prior art date
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- Granted
Links
- -1 acryloyl distamycin derivatives Chemical class 0.000 title claims description 61
- 229940100197 antimetabolite Drugs 0.000 title claims description 31
- 239000002256 antimetabolite Substances 0.000 title claims description 31
- 230000000340 anti-metabolite Effects 0.000 title claims description 30
- 206010028980 Neoplasm Diseases 0.000 title claims description 15
- 229940123237 Taxane Drugs 0.000 title claims description 6
- 238000002648 combination therapy Methods 0.000 title claims description 5
- 125000002924 primary amino group Chemical group [H]N([H])* 0.000 claims description 35
- 229940044684 anti-microtubule agent Drugs 0.000 claims description 26
- 125000002915 carbonyl group Chemical group [*:2]C([*:1])=O 0.000 claims description 23
- 239000008194 pharmaceutical composition Substances 0.000 claims description 16
- 238000000034 method Methods 0.000 claims description 14
- UPBAOYRENQEPJO-UHFFFAOYSA-N n-[5-[[5-[(3-amino-3-iminopropyl)carbamoyl]-1-methylpyrrol-3-yl]carbamoyl]-1-methylpyrrol-3-yl]-4-formamido-1-methylpyrrole-2-carboxamide Chemical group CN1C=C(NC=O)C=C1C(=O)NC1=CN(C)C(C(=O)NC2=CN(C)C(C(=O)NCCC(N)=N)=C2)=C1 UPBAOYRENQEPJO-UHFFFAOYSA-N 0.000 claims description 13
- GDTBXPJZTBHREO-UHFFFAOYSA-N bromine Chemical compound BrBr GDTBXPJZTBHREO-UHFFFAOYSA-N 0.000 claims description 12
- 229960005277 gemcitabine Drugs 0.000 claims description 12
- SDUQYLNIPVEERB-QPPQHZFASA-N gemcitabine Chemical compound O=C1N=C(N)C=CN1[C@H]1C(F)(F)[C@H](O)[C@@H](CO)O1 SDUQYLNIPVEERB-QPPQHZFASA-N 0.000 claims description 12
- 108010042747 stallimycin Proteins 0.000 claims description 12
- 241000124008 Mammalia Species 0.000 claims description 11
- 238000002560 therapeutic procedure Methods 0.000 claims description 11
- 230000000118 anti-neoplastic effect Effects 0.000 claims description 10
- 125000000217 alkyl group Chemical group 0.000 claims description 9
- 238000002360 preparation method Methods 0.000 claims description 9
- 239000003814 drug Substances 0.000 claims description 8
- 239000001257 hydrogen Substances 0.000 claims description 8
- 229910052739 hydrogen Inorganic materials 0.000 claims description 8
- 229910052794 bromium Inorganic materials 0.000 claims description 7
- 229910052801 chlorine Inorganic materials 0.000 claims description 7
- 150000003839 salts Chemical class 0.000 claims description 7
- 230000002195 synergetic effect Effects 0.000 claims description 7
- GHASVSINZRGABV-UHFFFAOYSA-N Fluorouracil Chemical compound FC1=CNC(=O)NC1=O GHASVSINZRGABV-UHFFFAOYSA-N 0.000 claims description 6
- VEXZGXHMUGYJMC-UHFFFAOYSA-N Hydrochloric acid Chemical compound Cl VEXZGXHMUGYJMC-UHFFFAOYSA-N 0.000 claims description 6
- 229930012538 Paclitaxel Natural products 0.000 claims description 6
- 229960001842 estramustine Drugs 0.000 claims description 6
- FRPJXPJMRWBBIH-RBRWEJTLSA-N estramustine Chemical compound ClCCN(CCCl)C(=O)OC1=CC=C2[C@H]3CC[C@](C)([C@H](CC4)O)[C@@H]4[C@@H]3CCC2=C1 FRPJXPJMRWBBIH-RBRWEJTLSA-N 0.000 claims description 6
- 229960002949 fluorouracil Drugs 0.000 claims description 6
- 229910052757 nitrogen Inorganic materials 0.000 claims description 6
- 125000004433 nitrogen atom Chemical group N* 0.000 claims description 6
- 229910052760 oxygen Inorganic materials 0.000 claims description 6
- 229960001592 paclitaxel Drugs 0.000 claims description 6
- 229910052717 sulfur Inorganic materials 0.000 claims description 6
- RCINICONZNJXQF-MZXODVADSA-N taxol Chemical compound O([C@@H]1[C@@]2(C[C@@H](C(C)=C(C2(C)C)[C@H](C([C@]2(C)[C@@H](O)C[C@H]3OC[C@]3([C@H]21)OC(C)=O)=O)OC(=O)C)OC(=O)[C@H](O)[C@@H](NC(=O)C=1C=CC=CC=1)C=1C=CC=CC=1)O)C(=O)C1=CC=CC=C1 RCINICONZNJXQF-MZXODVADSA-N 0.000 claims description 6
- UFHFLCQGNIYNRP-UHFFFAOYSA-N Hydrogen Chemical compound [H][H] UFHFLCQGNIYNRP-UHFFFAOYSA-N 0.000 claims description 5
- 125000001309 chloro group Chemical group Cl* 0.000 claims description 5
- 230000000694 effects Effects 0.000 claims description 5
- 208000032839 leukemia Diseases 0.000 claims description 5
- 125000001397 3-pyrrolyl group Chemical group [H]N1C([H])=C([*])C([H])=C1[H] 0.000 claims description 4
- 229940122803 Vinca alkaloid Drugs 0.000 claims description 4
- 125000003545 alkoxy group Chemical group 0.000 claims description 4
- NDMPLJNOPCLANR-UHFFFAOYSA-N 3,4-dihydroxy-15-(4-hydroxy-18-methoxycarbonyl-5,18-seco-ibogamin-18-yl)-16-methoxy-1-methyl-6,7-didehydro-aspidospermidine-3-carboxylic acid methyl ester Natural products C1C(CC)(O)CC(CC2(C(=O)OC)C=3C(=CC4=C(C56C(C(C(O)C7(CC)C=CCN(C67)CC5)(O)C(=O)OC)N4C)C=3)OC)CN1CCC1=C2NC2=CC=CC=C12 NDMPLJNOPCLANR-UHFFFAOYSA-N 0.000 claims description 3
- NMUSYJAQQFHJEW-KVTDHHQDSA-N 5-azacytidine Chemical compound O=C1N=C(N)N=CN1[C@H]1[C@H](O)[C@H](O)[C@@H](CO)O1 NMUSYJAQQFHJEW-KVTDHHQDSA-N 0.000 claims description 3
- 150000005727 5-fluoropyrimidines Chemical class 0.000 claims description 3
- GAGWJHPBXLXJQN-UORFTKCHSA-N Capecitabine Chemical compound C1=C(F)C(NC(=O)OCCCCC)=NC(=O)N1[C@H]1[C@H](O)[C@H](O)[C@@H](C)O1 GAGWJHPBXLXJQN-UORFTKCHSA-N 0.000 claims description 3
- GAGWJHPBXLXJQN-UHFFFAOYSA-N Capecitabine Natural products C1=C(F)C(NC(=O)OCCCCC)=NC(=O)N1C1C(O)C(O)C(C)O1 GAGWJHPBXLXJQN-UHFFFAOYSA-N 0.000 claims description 3
- UHDGCWIWMRVCDJ-CCXZUQQUSA-N Cytarabine Chemical compound O=C1N=C(N)C=CN1[C@H]1[C@@H](O)[C@H](O)[C@@H](CO)O1 UHDGCWIWMRVCDJ-CCXZUQQUSA-N 0.000 claims description 3
- MYMOFIZGZYHOMD-UHFFFAOYSA-N Dioxygen Chemical compound O=O MYMOFIZGZYHOMD-UHFFFAOYSA-N 0.000 claims description 3
- ZDZOTLJHXYCWBA-VCVYQWHSSA-N N-debenzoyl-N-(tert-butoxycarbonyl)-10-deacetyltaxol Chemical compound O([C@H]1[C@H]2[C@@](C([C@H](O)C3=C(C)[C@@H](OC(=O)[C@H](O)[C@@H](NC(=O)OC(C)(C)C)C=4C=CC=CC=4)C[C@]1(O)C3(C)C)=O)(C)[C@@H](O)C[C@H]1OC[C@]12OC(=O)C)C(=O)C1=CC=CC=C1 ZDZOTLJHXYCWBA-VCVYQWHSSA-N 0.000 claims description 3
- IVTVGDXNLFLDRM-HNNXBMFYSA-N Tomudex Chemical compound C=1C=C2NC(C)=NC(=O)C2=CC=1CN(C)C1=CC=C(C(=O)N[C@@H](CCC(O)=O)C(O)=O)S1 IVTVGDXNLFLDRM-HNNXBMFYSA-N 0.000 claims description 3
- JXLYSJRDGCGARV-WWYNWVTFSA-N Vinblastine Natural products O=C(O[C@H]1[C@](O)(C(=O)OC)[C@@H]2N(C)c3c(cc(c(OC)c3)[C@]3(C(=O)OC)c4[nH]c5c(c4CCN4C[C@](O)(CC)C[C@H](C3)C4)cccc5)[C@@]32[C@H]2[C@@]1(CC)C=CCN2CC3)C JXLYSJRDGCGARV-WWYNWVTFSA-N 0.000 claims description 3
- 230000003432 anti-folate effect Effects 0.000 claims description 3
- 229940127074 antifolate Drugs 0.000 claims description 3
- 239000002246 antineoplastic agent Substances 0.000 claims description 3
- 229960002756 azacitidine Drugs 0.000 claims description 3
- 229960004117 capecitabine Drugs 0.000 claims description 3
- 125000004093 cyano group Chemical group *C#N 0.000 claims description 3
- 229960000684 cytarabine Drugs 0.000 claims description 3
- UHDGCWIWMRVCDJ-ZAKLUEHWSA-N cytidine Chemical class O=C1N=C(N)C=CN1[C@H]1[C@H](O)[C@@H](O)[C@H](CO)O1 UHDGCWIWMRVCDJ-ZAKLUEHWSA-N 0.000 claims description 3
- 229960003668 docetaxel Drugs 0.000 claims description 3
- 229960000961 floxuridine Drugs 0.000 claims description 3
- ODKNJVUHOIMIIZ-RRKCRQDMSA-N floxuridine Chemical compound C1[C@H](O)[C@@H](CO)O[C@H]1N1C(=O)NC(=O)C(F)=C1 ODKNJVUHOIMIIZ-RRKCRQDMSA-N 0.000 claims description 3
- 239000004052 folic acid antagonist Substances 0.000 claims description 3
- 125000005842 heteroatom Chemical group 0.000 claims description 3
- 125000004435 hydrogen atom Chemical group [H]* 0.000 claims description 3
- 239000002502 liposome Substances 0.000 claims description 3
- 239000001301 oxygen Substances 0.000 claims description 3
- 125000000843 phenylene group Chemical group C1(=C(C=CC=C1)*)* 0.000 claims description 3
- 229960004432 raltitrexed Drugs 0.000 claims description 3
- 229920006395 saturated elastomer Polymers 0.000 claims description 3
- 125000004434 sulfur atom Chemical group 0.000 claims description 3
- 229960001674 tegafur Drugs 0.000 claims description 3
- WFWLQNSHRPWKFK-ZCFIWIBFSA-N tegafur Chemical compound O=C1NC(=O)C(F)=CN1[C@@H]1OCCC1 WFWLQNSHRPWKFK-ZCFIWIBFSA-N 0.000 claims description 3
- NOYPYLRCIDNJJB-UHFFFAOYSA-N trimetrexate Chemical compound COC1=C(OC)C(OC)=CC(NCC=2C(=C3C(N)=NC(N)=NC3=CC=2)C)=C1 NOYPYLRCIDNJJB-UHFFFAOYSA-N 0.000 claims description 3
- 229960001099 trimetrexate Drugs 0.000 claims description 3
- 229960003048 vinblastine Drugs 0.000 claims description 3
- JXLYSJRDGCGARV-XQKSVPLYSA-N vincaleukoblastine Chemical compound C([C@@H](C[C@]1(C(=O)OC)C=2C(=CC3=C([C@]45[C@H]([C@@]([C@H](OC(C)=O)[C@]6(CC)C=CCN([C@H]56)CC4)(O)C(=O)OC)N3C)C=2)OC)C[C@@](C2)(O)CC)N2CCC2=C1NC1=CC=CC=C21 JXLYSJRDGCGARV-XQKSVPLYSA-N 0.000 claims description 3
- 229960004355 vindesine Drugs 0.000 claims description 3
- UGGWPQSBPIFKDZ-KOTLKJBCSA-N vindesine Chemical compound C([C@@H](C[C@]1(C(=O)OC)C=2C(=CC3=C([C@]45[C@H]([C@@]([C@H](O)[C@]6(CC)C=CCN([C@H]56)CC4)(O)C(N)=O)N3C)C=2)OC)C[C@@](C2)(O)CC)N2CCC2=C1N=C1[C]2C=CC=C1 UGGWPQSBPIFKDZ-KOTLKJBCSA-N 0.000 claims description 3
- 229960002066 vinorelbine Drugs 0.000 claims description 3
- GBABOYUKABKIAF-GHYRFKGUSA-N vinorelbine Chemical compound C1N(CC=2C3=CC=CC=C3NC=22)CC(CC)=C[C@H]1C[C@]2(C(=O)OC)C1=CC([C@]23[C@H]([C@]([C@H](OC(C)=O)[C@]4(CC)C=CCN([C@H]34)CC2)(O)C(=O)OC)N2C)=C2C=C1OC GBABOYUKABKIAF-GHYRFKGUSA-N 0.000 claims description 3
- 208000003174 Brain Neoplasms Diseases 0.000 claims description 2
- 206010027476 Metastases Diseases 0.000 claims description 2
- 229940034982 antineoplastic agent Drugs 0.000 claims description 2
- 210000000481 breast Anatomy 0.000 claims description 2
- 210000001072 colon Anatomy 0.000 claims description 2
- 201000010099 disease Diseases 0.000 claims description 2
- 208000037265 diseases, disorders, signs and symptoms Diseases 0.000 claims description 2
- 239000003937 drug carrier Substances 0.000 claims description 2
- 125000004356 hydroxy functional group Chemical group O* 0.000 claims description 2
- 210000003734 kidney Anatomy 0.000 claims description 2
- 210000004185 liver Anatomy 0.000 claims description 2
- 210000004072 lung Anatomy 0.000 claims description 2
- 201000001441 melanoma Diseases 0.000 claims description 2
- 230000009401 metastasis Effects 0.000 claims description 2
- 230000014399 negative regulation of angiogenesis Effects 0.000 claims description 2
- 230000001613 neoplastic effect Effects 0.000 claims description 2
- 210000001672 ovary Anatomy 0.000 claims description 2
- 210000000496 pancreas Anatomy 0.000 claims description 2
- 239000000546 pharmaceutical excipient Substances 0.000 claims description 2
- 230000002265 prevention Effects 0.000 claims description 2
- 210000002784 stomach Anatomy 0.000 claims description 2
- 229960004528 vincristine Drugs 0.000 claims description 2
- OGWKCGZFUXNPDA-XQKSVPLYSA-N vincristine Chemical compound C([N@]1C[C@@H](C[C@]2(C(=O)OC)C=3C(=CC4=C([C@]56[C@H]([C@@]([C@H](OC(C)=O)[C@]7(CC)C=CCN([C@H]67)CC5)(O)C(=O)OC)N4C=O)C=3)OC)C[C@@](C1)(O)CC)CC1=C2NC2=CC=CC=C12 OGWKCGZFUXNPDA-XQKSVPLYSA-N 0.000 claims description 2
- OGWKCGZFUXNPDA-UHFFFAOYSA-N vincristine Natural products C1C(CC)(O)CC(CC2(C(=O)OC)C=3C(=CC4=C(C56C(C(C(OC(C)=O)C7(CC)C=CCN(C67)CC5)(O)C(=O)OC)N4C=O)C=3)OC)CN1CCC1=C2NC2=CC=CC=C12 OGWKCGZFUXNPDA-UHFFFAOYSA-N 0.000 claims description 2
- FBOZXECLQNJBKD-ZDUSSCGKSA-N L-methotrexate Chemical compound C=1N=C2N=C(N)N=C(N)C2=NC=1CN(C)C1=CC=C(C(=O)N[C@@H](CCC(O)=O)C(O)=O)C=C1 FBOZXECLQNJBKD-ZDUSSCGKSA-N 0.000 claims 2
- 229960000485 methotrexate Drugs 0.000 claims 2
- 125000000981 3-amino-3-oxopropyl group Chemical group [H]C([*])([H])C([H])([H])C(=O)N([H])[H] 0.000 claims 1
- 239000004480 active ingredient Substances 0.000 claims 1
- ZAAQBUWPDFZWFW-UHFFFAOYSA-N n-(3-amino-3-iminopropyl)-4-[[4-[[4-(2-bromoprop-2-enoylamino)-1-methylpyrrole-2-carbonyl]amino]-1-methylpyrrole-2-carbonyl]amino]-1-methylpyrrole-2-carboxamide;hydrochloride Chemical compound Cl.C1=C(C(=O)NCCC(N)=N)N(C)C=C1NC(=O)C1=CC(NC(=O)C=2N(C=C(NC(=O)C(Br)=C)C=2)C)=CN1C ZAAQBUWPDFZWFW-UHFFFAOYSA-N 0.000 claims 1
- IPKHXIAPPAKHTB-UHFFFAOYSA-N 2-[[4-[[4-[[4-[[4-(2-bromoprop-2-enoylamino)-1-methylpyrrole-2-carbonyl]amino]-1-methylpyrrole-2-carbonyl]amino]-1-methylpyrrole-2-carbonyl]amino]-1-methylpyrrole-2-carbonyl]amino]ethyl-(diaminomethylidene)azanium;chloride Chemical compound Cl.C1=C(C(=O)NCCN=C(N)N)N(C)C=C1NC(=O)C1=CC(NC(=O)C=2N(C=C(NC(=O)C=3N(C=C(NC(=O)C(Br)=C)C=3)C)C=2)C)=CN1C IPKHXIAPPAKHTB-UHFFFAOYSA-N 0.000 description 6
- 150000001875 compounds Chemical class 0.000 description 6
- 230000000259 anti-tumor effect Effects 0.000 description 5
- 239000000203 mixture Substances 0.000 description 5
- 125000003739 carbamimidoyl group Chemical group C(N)(=N)* 0.000 description 4
- 229940079593 drug Drugs 0.000 description 3
- 229920000128 polypyrrole Polymers 0.000 description 3
- WKBOTKDWSSQWDR-UHFFFAOYSA-N Bromine atom Chemical compound [Br] WKBOTKDWSSQWDR-UHFFFAOYSA-N 0.000 description 2
- ZAMOUSCENKQFHK-UHFFFAOYSA-N Chlorine atom Chemical compound [Cl] ZAMOUSCENKQFHK-UHFFFAOYSA-N 0.000 description 2
- 241001529936 Murinae Species 0.000 description 2
- 241000699670 Mus sp. Species 0.000 description 2
- 230000000719 anti-leukaemic effect Effects 0.000 description 2
- 201000011510 cancer Diseases 0.000 description 2
- 239000000460 chlorine Substances 0.000 description 2
- 238000009472 formulation Methods 0.000 description 2
- 229950009902 stallimycin Drugs 0.000 description 2
- 230000004083 survival effect Effects 0.000 description 2
- JOXIMZWYDAKGHI-UHFFFAOYSA-N toluene-4-sulfonic acid Chemical compound CC1=CC=C(S(O)(=O)=O)C=C1 JOXIMZWYDAKGHI-UHFFFAOYSA-N 0.000 description 2
- 231100000331 toxic Toxicity 0.000 description 2
- 230000002588 toxic effect Effects 0.000 description 2
- 231100000419 toxicity Toxicity 0.000 description 2
- 230000001988 toxicity Effects 0.000 description 2
- COVZYZSDYWQREU-UHFFFAOYSA-N Busulfan Chemical compound CS(=O)(=O)OCCCCOS(C)(=O)=O COVZYZSDYWQREU-UHFFFAOYSA-N 0.000 description 1
- 241000699666 Mus <mouse, genus> Species 0.000 description 1
- 239000003242 anti bacterial agent Substances 0.000 description 1
- 230000001946 anti-microtubular Effects 0.000 description 1
- 230000000842 anti-protozoal effect Effects 0.000 description 1
- 230000000840 anti-viral effect Effects 0.000 description 1
- 230000003115 biocidal effect Effects 0.000 description 1
- 125000002837 carbocyclic group Chemical group 0.000 description 1
- SFZULDYEOVSIKM-UHFFFAOYSA-N chembl321317 Chemical compound C1=CC(C(=N)NO)=CC=C1C1=CC=C(C=2C=CC(=CC=2)C(=N)NO)O1 SFZULDYEOVSIKM-UHFFFAOYSA-N 0.000 description 1
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- 125000001495 ethyl group Chemical group [H]C([H])([H])C([H])([H])* 0.000 description 1
- 125000000623 heterocyclic group Chemical group 0.000 description 1
- 230000005917 in vivo anti-tumor Effects 0.000 description 1
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- 238000007918 intramuscular administration Methods 0.000 description 1
- 238000001990 intravenous administration Methods 0.000 description 1
- 125000000959 isobutyl group Chemical group [H]C([H])([H])C([H])(C([H])([H])[H])C([H])([H])* 0.000 description 1
- 125000001449 isopropyl group Chemical group [H]C([H])([H])C([H])(*)C([H])([H])[H] 0.000 description 1
- 239000002207 metabolite Substances 0.000 description 1
- 125000000956 methoxy group Chemical group [H]C([H])([H])O* 0.000 description 1
- 125000002496 methyl group Chemical group [H]C([H])([H])* 0.000 description 1
- 150000007522 mineralic acids Chemical class 0.000 description 1
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- QJGQUHMNIGDVPM-UHFFFAOYSA-N nitrogen group Chemical group [N] QJGQUHMNIGDVPM-UHFFFAOYSA-N 0.000 description 1
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Description
COMBINED THERAPY AGAINST TUMORS COMPRISING SUBSTITUTED ACRYLOYL DISTAMYCIN DERIVATIVES, TAXANES AND/OR ANTIMETABOLITES
The present invention relates to the field of cancer treatment and provides an antitumor composition comprising a substituted acryloyl distamycin derivative, more particularly an α-bromo- or α-chloro-acryloyl distamycin derivative, an antimicrotubule agent and/or an antimetabolite, having a synergistic antineoplastic effect.
Distamycin A and analogues thereof, hereinafter referred to as distamycin and distamycin-like derivatives, are known in the art as cytotoxic agents useful in antitumor therapy.
Distamycin A is an antibiotic substance with antiviral and antiprotozoal activity, having a polypyrrole framework [Nature 203: 1064 (1964); J. Med. Chem. 32: 774-778 (1989)]. The international patent applications WO 90/11277, WO 98/04524, WO 98/21202, WO 99/50265, WO 99/50266 and WO 01/40181 (claiming priority from British patent application No. 9928703.9), all in the name of the applicant itself and herewith incorporated by reference, disclose acryloyl distamycin derivatives wherein the amidino moiety of distamycin is optionally replaced by nitrogen-containing ending groups such as, for instance, cyanamidino, N-methylamidino, guanidino, carbamoyl, amidoxime, cyano and the like, and/or wherein the polypyrrole framework of distamycin, or part of it, is replaced by varying carbocyclic or heterocyclic moieties.
The present invention provides, in a first aspect, a pharmaceutical composition for use in antineoplastic therapy in mammals, including humans, comprising a pharmaceutically acceptable carrier or excipient; an acryloyl distamycin derivative of formula (I):
wherein:
Ri is a bromine or chlorine atom;
R2 is a distamycin or distamycin-like framework; or a pharmaceutically acceptable salt thereof; and an antimicrotubule agent and/or an antimetabolite.
The present invention includes, within its scope, the pharmaceutical compositions comprising any of the possible isomers covered by the compounds of formula (I), both considered separately or in admixture, as well as the metabolites and the pharmaceutically acceptable bio-precursors (otherwise known as pro-drugs) of the compounds of formula (I) .
In the present description, unless otherwise specified, with the term distamycin or distamycin-like framework R2 we intend any moiety structurally closely related to distamycin itself, for instance by optionally replacing the ending amidino moiety of distamycin and/or its polypyrrole framework, or part of it.
Antimicrotubule agents and antimetabolites are widely known in the art as antitumor agents; see, for a general reference, Cancer, Principles and Practice of Oncology, Lippincort-Raven Ed. (1997), 432-452 and 467-483
According to a preferred embodiment of the invention, herewith provided are the above pharmaceutical compositions wherein the antimicrotubule agents are, for instance, taxanes, e.g. paclitaxel or docetaxel; vinca alkaloids, e.g. vincristine, vinblastine, vindesine, vinorelbine; and estramustine, optionally encapsulated within liposomes. Preferred antimetabolites are, for instance, antifolates, e.g. metotrexate, trimetrexate, tomudex; 5-fluoropyrimidines, e.g. 5-FU, floxuridine, ftorafur and capecitabine; cytidine analogs, e.g. cytarabine, azacitidine and gemcitabine.
Particularly preferred antimicrotubule agents are paclitaxel and estramustine whereas preferred antimetabolites are 5-fluorouracil or gemcitabine.
According to another preferred embodiment of the invention, herewith provided are the
above pharmaceutical compositions wherein, within the acryloyl distamycin derivative of formula (I), R has the above reported meanings and R is a group of formula (H) below:
wherein m is an integer from 0 to 2; n is an integer from 2 to 5; r is O or 1;
X and Y are, the same or different and independently for each heterocycUc ring, a nitrogen atom or a CH group;
G is phenylene, a 5 or 6 membered saturated or unsaturated heterocycUc ring with from 1 to 3 heteroatoms selected among N, O or S, or it is a group of formula (J_H) below:
wherein Q is a nitrogen atom or a CH group and W is an oxygen or sulfur atom or it is a group NR wherein R3 is hydrogen or Cj-C4 alkyl; B is selected from the group consisting of
CN NR5R6 — CONR5R6 — NHCONR5R6
wherein R is cyano, amino, hydroxy or Cι-C4 alkoxy; R5, R6 and R7, the same or different, are hydrogen or -C4 alkyl.
hi the present description, unless otherwise specified, with the term Cι-C alkyl or alkoxy group we intend a straight or branched group selected from methyl, ethyl, n- propyl, isopropyl, n-butyl, isobutyl, sec-butyl, tert-butyl, methoxy. ethoxy, n-propoxy, isopropoxy, n-butoxy, isobutoxy, sec-butoxy or tert-butoxy.
Even more preferred are the pharmaceutical compositions of the invention comprising the above acryloyl distamycin derivative of formula (I) wherein Ri is bromine or chlorine; R2 is the above group of formula (TJ) wherem r is 0, m is 0 or 1, n is 4 and B has the above reported meanings. Still more preferred, within this class, are the pharmaceutical compositions comprising the compounds of formula (I) wherein Ri is bromine or chlorine; R2 is the above group of formula (II) wherein r is 0, m is 0 or 1, n is 4, X and Y are both CH groups and B is selected from:
— CN . — CONR5R6 — NHCONR5R6
' ; wherein R4 is cyano or hydroxy and R5, Re and R7, the same or different, are hydrogen or Cι-C alkyl.
Pharmaceutically acceptable salts of the compounds of formula (I) are those with pharmaceutically acceptable inorganic or organic acids such as, for instance, hydrochloric, hydrobromic, sulfuric, nitric, acetic, propionic, succinic, malonic, citric, tartaric, methanesulfonic, p-toluenesulfonic acid and the like.
Examples of preferred acryloyl distamycin derivatives of formula (I), within the compositions object of the invention, optionally in the form of pharmaceutically acceptable salts, preferably with hydrochloric acid, are:
1. N-(5- {[(5- {[(5- {[(2- {[amino(imino)methyl]amino} ethyl)amino]carbonyl} -1 - methyl- 1 H-pyrrol-3 -yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 -
yl)amino jcarbonyl} - 1 -methyl- lH-pyrrol-3-yl)-4-[(2-bromoacryloyl)amino]- 1 - methyl-lH-pyrrole-2-carboxamide hydrochloride;
2. N-(5-{[(5-{[(5-{[(2-{[amino(imino)methyl]ammo}propyl)amino]carbonyl}-l- methyl- 1 H-pyrrol-3 -yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 - yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 -yl)-4- [(2-bromoacryloyl)amino]- 1 - methyl-lH-pyrrole-2-carboxamide hydrochloride;
3. N-(5-{[(5-{[(5-{[(3-amino-3-iminopropyl)amino]carbonyl}-l-methyl-lH-pyrrol-3- yl)amino]carbonyl} - 1 -methyl- lH-pyrrol-3-yl)amino]carbonyl} - 1 -methyl- 1 H- pyrrol-3 -yl)-4- [(2-bromoacryloyl)amino] - 1 -methyl- 1 H-pyrrole-2-carboxamide hydrochloride;
4. N-(5- { [(5 - { [(5 - { [(3 -amino-3 -iminopropyl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 - yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 -yl)amino] carbonyl} - 1 -methyl- 1 H- pyrrol-3 -yl)-4- [(2-bromoacryloyl)amino] - 1 -methyl- 1 H-imidazole-2-carboxamide hydrochloride; 5. N-(5-{[(5-{[(5-{[(3 -amino-3 -iminopropyl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 - yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 -yl)amino] carbonyl} - 1 -methyl- 1 H- pyrrol-3-yl)-3-[(2-bromoacryloyl)amino]-l-methyl-lH-pyrazole-5-carboxamide hydrochloride;
6. N-(5-{[(5-{[(5-{[(3-amino-3-oxopropyl)amino]carbonyl}-l-methyl-lH-pyrrol-3- yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3-yl)amino] carbonyl} - 1 -methyl- 1 H- pyrrol-3-yl)-3-[(2-bromoacryloyl)amino]-l-methyl-lH-pyrazole-5-carboxamide;
7. N-(5-{[(5-{[(5-{[(2-{ [amino(imino)methyl] amino } ethyl)amino] carbonyl} - 1 - methyl- 1 H-pyrrol-3 -yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 - yl)amino]carbonyl}-l-methyl-lH-pyrrol-3-yl)-4-[(2-chloroacryloyl)amino]-l- methyl- 1 H-pyrrole-2-carboxamide hydrochloride;
8. N-(5- { [(5- { [(3 - {[amino(imino)methyl] amino}propyl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 -yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 -yl)-4- [(2- bromoacryloyl)amino] - 1 -methyl- 1 H-p yrrole-2-carboxamide hydrochloride;
9. N-(5-{[(5-{[(3-ammo-3-iminopropyl)amino]carbonyl}-l-methyl-lH-pyrrol-3- yl)amino]carbonyl}-l-methyl-lH-pyrrol-3-yl)-4-[(2-bromoacryloyl)amino]-l- methyl-lH-pyrrole-2-carboxamide hydrochloride; and
10. N-{5-[({5-[({5-[({3-[(aminocarbonyl)amino]propyl}amino)carbonyl]-l-ιτιethyl- 1 H-pyrrol-3 -yl } amino)carbonyl] - 1 -methyl- 1 H-pyrrol-3 -yl} amino)carbonyl] - 1 - methyl- 1 H-pyrrol-3 -yl} -4- [(2-bromoacryloyl)amino] - 1 -methyl- 1 H-pyrrole-2- carboxamide.
The above compounds of formula (I), either specifically identified as such or by means of the general formula, are known or easily prepared according to known methods as reported, for instance, in the aforementioned international patent applications WO 90/11277, WO 98/04524, WO 98/21202, WO 99/50265 and WO 99/50266 and WO 01/40181.
The present invention further provides a product comprising an acryloyl distamycin derivative of formula (I), as defined above, an antimicrotubule agent and/or an antimetabolite, as a combined preparation for simultaneous, separate or sequential use in antitumor therapy.
Particularly preferred, in this respect, is a product comprising N-(5-{[(5-{[(5-{[(2- [amino(imino)methyl]amino}ethyl)amino]carbonyl}-l-methyl-lH-pyrrol-3- yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 -yl)amino] carbonyl} - 1 -methyl- lH-pyrrol-3 - yl)-4-[(2-bromoacryloyl)amino]-l-methyl-lH-pyrrole-2-carboxamide hydrochloride (internal code PNU 166196) and gemcitabine, as a combined preparation for simultaneous, separate or sequential use in antitumor therapy.
A further aspect of the present invention is to provide a method of treating a mammal, including humans, suffering from a neoplastic disease state, which method comprises administering to said mammal the above acryloyl distamycin derivative of formula (I), an antimicrotubule agent and/or an antimetabolite, in amounts effective to produce a synergistic antineoplastic effect.
The present invention also provides a method for lowering the side effects caused by antineoplastic therapy with an antineoplastic agent in a mammal in need thereof,
including humans, the method comprising administering to said mammal a combined preparation comprising an acryloyl distamycin derivative of formula (I), an antimicrotubule agent and/or an antimetabolite, in amounts effective to produce a synergistic antineoplastic effect. By the term "synergistic antineoplastic effect", as used herein, it is meant the inhibition of the growth tumor, preferably the complete regression of the tumor, by administering an effective amount of the combination comprising an acryloyl distamycin derivative of formula (I), an antimicrotubule agent and/or an antimetabolite to mammals, including humans. By the term "admimstered " or "administering", as used herein, it is meant parenteral and/or oral administration; the term "parenteral" means intravenous, subcutaneous and intramuscular administration.
In the method of the present invention, the acryloyl distamycin derivative may be admimstered simultaneously with the antimicrotubule agent or with the antimetabolite.
Alternatively, the two drugs may be administered sequentially in either order.
When the acryloyl distamycin derivative is administered with both the antimicrotubule agent and the antimetabolite, according to an embodiment of the invention, the drugs are preferably administered sequentially, in any order. In this respect, it will be appreciated that the actual preferred method and order of administration will vary according to, inter alias, the particular formulation of the acryloyl distamycin of formula (I) being used, the particular formulation of the antimicrotubule agent and/or the antimetabolite being used, the particular tumor model being treated as well as the particular host being treated. To administer the acryloyl distamycin derivative of formula (I), according to the method of the invention, the course of therapy generally employed comprises doses varying from about 0.05 to about 100 mg/m2 of body surface area and, more preferably, from about 0J to about 50 mg/m2 of body surface area.
For the admimstration of the taxanes, according to the method of the invention, the course of therapy generally employed comprises doses varying from about 1 to about
1000 mg/m2 of body surface area and, more preferably, from about 10 to about 500 mg/m2 of body surface area.
For the administration of the vinca alkaloids, according to the method of the invention, the course of therapy generally employed comprises doses varying from about 0J to about 1000 mg/m2 of body surface area and, more preferably, from about 0.5 to about 100 mg/m of body surface area.
For the administration of the antimetabolite according to the invention, the course of therapy generally employed comprises doses varying from about 0J to about 10 g/m2 of body surface area and, more preferably, from about 1 to about 5 g/m2 of body surface area.
The antineoplastic therapy of the present invention is particularly suitable for treating breast, ovary, lung, colon, kidney, stomach, pancreas, liver, melanoma, leukemia and brain tumors in mammals, including humans.
In a further aspect, the present invention is directed to a composition comprising an effective amount of an acryloyl distamycin derivative of formula (I), as defined above, an antimicrotubule agent and/or an antimetabolite, in the preparation of a medicament for use in the prevention or treatment of metastasis or in the treatment of tumors by inhibition of angiogenesis.
As stated above, the effect of an acryloyl distamycin derivative of formula (I) with an antimicrotubule agent and/or an antimetabolite, is significantly increased without a parallel increase of toxicity. In other words, the combined therapy of the present invention enhances the antitumoral effects of the acryloyl distamycin derivative and of the other drug, being either an antimicrotubule, an antimetabolite or a combination thereof and, hence, provides the most effective and least toxic treatment for tumors.
The superadditive effects of the combined preparations of the invention are shown, for instance, by the following in vivo antitumor activity data which are intended to illustrate the present invention without posing any limitation to it.
Table 1 shows the antileukemic activity on disseminated L1210 murine leukemia obtained by combining N-(5-{[(5-{[(5-{[(2-
{ [amino(imino)methyl] amino } ethyl)amino] carbonyl } - 1 -methyl- 1 H-pyrrol-3 - yl)amino]carbonyl} - 1 -methyl- lH-pyrrol-3-yl)amino]carbonyl} - 1 -methyl- lH-pyrrol-3- yl)-4- [(2-bromoacryloyl)amino] - 1 -methyl- 1 H-pyrrole-2-carboxamide hydrochloride, as a representative compound of formula (I) - internal code PNU 166196, with gemcitabine.
At the dose of 15 mg/kg of gemcitabine alone (day +1 after tumor injection, 2 h after PNU 166196 administration) and at the dose of 0J mg/kg of PNU 166196 alone (days +1,6) were associated, without toxicity, ILS% values of 50 and 58, respectively. Combining gemcitabine and PNU 166196 at the same doses with the same schedule, an increase of activity with T_LS% values of 127 were observed, thus indicating a synergistic effect.
Table 1: Antileukemic activity against disseminated L12101 murine leukemia of an acryloyl distamycin derivative (I) in combination with gemcitabine.
1) L1210 leukemia cells (105/mouse) are injected iv on day 0.
2) Treatment is given starting on day 1 after tumor transplantation (day 0).
3) Increase in life span: [(median survival time of treated mice/median survival time of controls)x 100]- 100
4) Number of toxic deaths/number of mice.
(*) treatment 2 h after PNU 166196 administration.
Claims (25)
1. A pharmaceutical composition comprising a pharmaceutically acceptable carrier or excipient and, as active ingredient, an acryloyl distamycin derivative of formula (I):
wherein:
Ri is a bromine or chlorine atom;
R is a distamycin or distamycin-like framework; or a pharmaceutically acceptable salt thereof; and an antimicrotubule agent and/or an antimetabolite.
2. A pharmaceutical composition according to claim 1 wherein the antimicrotubule agent is selected from taxanes, including paclitaxel and docetaxel; vinca alkaloids, including vmcristine, vinblastine, vindesine, vinorelbine; and estramustine, optionally encapsulated within liposomes.
3. A pharmaceutical composition according to claim 2 wherein the antimicrotubule agent is paclitaxel or estramustine.
4. A pharmaceutical composition according to claim 1 wherein the antimetabolite is selected from antifolates, including methotrexate, tomudex and trimetrexate; 5- fluoropyrimidine derivatives, including 5-fluorouracil, floxuridine, ftorafur and capecitabine; and cytidine analogs, including cytarabine, azacitidine and gemcitabine.
5. A pharmaceutical composition according to claim 4 wherein the antimetabolite is selected from 5-fluorouracil or gemcitabine.
6. A pharmaceutical composition according to claim 1 comprising an acryloyl distamycin derivative of formula (I)
wherein:
Ri is a bromine or chlorine atom;
R2 is a group of formula (11)
wherein m is an integer from 0 to 2; n is an integer from 2 to 5; r is O or 1;
X and Y are, the same or different and independently for each heterocycUc ring, a nitrogen atom or a CH group;
G is phenylene, a 5 or 6 membered saturated or unsaturated heterocycUc ring with from 1 to 3 heteroatoms selected among N, O or S, or it is a group of formula (ID) below:
wherein Q is a nitrogen atom or a CH group and W is an oxygen or sulfur atom or it is a group NR3 wherein R3 is hydrogen or Cι-C alkyl; B is selected from the group consisting of
— CN . -NR5R6 . - - CONR5R6 — NHCONR5R6
wherein R is cyano, amino, hydroxy or Cι-C4 alkoxy; R5, R6 and R7, the same or different, are hydrogen or Cι-C alkyl.
7. A pharmaceutical composition according to claim 6 comprising an acryloyl distamycin derivative of formula (I) wherein Ri, R2 and B are as defined in claim 6, r is 0, m is 0 or 1 and n is 4.
8. A pharmaceutical composition according to claim 7 comprising an acryloyl distamycin derivative of formula (I) wherein Ri and R are as defined in claim 6, r is 0, m is 0 or 1, n is 4, X and Y are both CH groups and B is selected from:
— CN . — CONR5R6 — NHCONR5R6 i wherein R-j is cyano or hydroxy and R5, R6 and R7, the same or different, are hydrogen or Cι-C4 alkyl.
9. A pharmaceutical composition according to claim 1 comprising an acryloyl distamycin derivative, optionally in the form of a pharmaceutically acceptable salt, selected from the group consisting of:
1. N-(5- { [(5- { [(5- { [(2- { [amino(imino)methyl] amino} ethyl)amino] carbonyl} - 1 - methyl-lH-pyrrol-3-yl)amino]carbonyl}-l-methyl-lH-pyrrol-3-
yl)amino]carbonyl}-l-methyl-lH-pyrrol-3-yl)-4-[(2-bromoacryloyl)amino]-l- methyl- 1 H-pyrrole-2-carboxamide hydrochloride;
2. N-(5- {[(5- {[(5- {[(2- {[amino(imino)methyl]amino}propyl)amino]carbonyl} -1 - methyl-lH-pyrrol-3-yl)amino]carbonyl}-l-methyl-lH-pyrrol-3- yl)amino]carbonyl}-l-methyl-lH-pyrrol-3-yl)-4-[(2-bromoacryloyl)amino]-l- methyl- 1 H-pyrrole-2-carboxamide hydrochloride;
3. N-(5-{[(5-{[(5-{[(3-ammo-3-iminopropyl)amino]carbonyl}-l-methyl-lH-pyrrol-3- yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 -yl)amino] carbonyl} - 1 -methyl- 1 H- pyrrol-3 -yl)-4- [(2-bromoacryloyl)amino] - 1 -methyl- 1 H-pyrrole-2-carboxamide hydrochloride;
4. N-(5-{[(5-{[(5-{[(3-amino-3-iminopropyl)amino]carbonyl}-l-methyl-lH-pyrrol-3- yl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 -yl)amino] carbonyl} - 1 -methyl- 1H- pyι ol-3-yl)-4-[(2-bromoacιyloyl)amino]-l-memyl-lH-imidazole-2-carboxamide hydrochloride; 5. N-(5-{[(5-{[(5-{[(3-amino-3-iminopropyl)amino]carbonyl}-l-methyl-lH-pyrrol-3- yl)amino] carbonyl} -1 -methyl- 1 H-pyrrol-3 -yl)amino] carbonyl} - 1 -methyl- 1 H- pyrrol-3-yl)-3-[(2-bromoacryloyl)amino]-l-methyl-lH-pyrazole-5-carboxamide hydrochloride;
6. N-(5-{[(5-{[(5-{[(3-amino-3-oxopropyl)amino]carbonyl}-l-methyl-lH-pyπOl-3- yl)amino]carbόnyl} - 1 -methyl- 1 H-pyrrol-3-yl)amino]carbonyl} - 1 -methyl- 1H- pyrrol-3 -yl)-3 - [(2-bromoacryloyl)amino] - 1 -methyl- 1 H-pyrazole-5-carboxamide;
7. N-(5-{[(5-{[(5-{[(2-{[amino(imino)methyl]amino}ethyl)amino]carbonyl}-l- methyl- 1 H-pyrrol-3 -yl)arnino] carbonyl} - 1 -methyl- 1 H-pyrrol-3 - yl)amino]carbonyl}-l-methyl-lH-pyrrol-3-yl)-4-[(2-chloroacryloyl)amino]-l- methyl- 1 H-pyrrole-2-carboxamide hydrochloride;
8. N-(5-{[(5-{[(3-{ [amino(imino)methyl] amino } propyl)amino] carbonyl} - 1 -methyl- 1 H-pyrrol-3-yl)amino]carbonyl} - 1 -methyl- 1 H-pyrrol-3-yl)-4-[(2- bromoacryloyl)amino]-l-methyl-lH-pyrrole-2-carboxamide hydrochloride;
9. N-(5-{[(5-{[(3-amino-3-iminopropyl)amino]carbonyl}-l-methyl-lH-pyrrol-3- yl)amino]carbonyl}-l-methyl-lH-pyrrol-3-yl)-4-[(2-bromoacryloyl)amino]-l- methyl-lH-pyrrole-2-carboxamide hydrochloride; and
10. N- {5-[( {5-[( {5-[( {3-[(aminocarbonyl)amino]propyl} amino)carbonyl]- 1 -methyl- 1 H-pyrrol-3 -yl} amino)carbonyl] - 1 -methyl- 1 H-pyrrol-3 -yl} amino)carbonyl] - 1 - methyl- 1 H-pyrrol-3 -yl} -4- [(2-bromoacryloyl)amino] - 1 -methyl- 1 H-pyrrole-2- carboxamide.
10. Products comprising an acryloyl distamycin derivative of formula (I):
wherein:
Ri is a bromine or chlorine atom; R2 is a distamycin or distamycin-like framework; or a pharmaceutically acceptable salt thereof; an antimicrotubule agent and/or an antimetabolite, as a combined preparation for simultaneous, separate or sequential use in the treatment of tumors.
11. Products according to claim 10 wherein the antimicrotubule agent is selected from taxanes, including paclitaxel and docetaxel; vinca alkaloids, including vincristine, vinblastine, vindesine, vinorelbine; and estramustine, optionally encapsulated within liposomes.
12. Products according to claim 11 wherein the antimicrotubule agent is paclitaxel or estramustine.
13. Products according to claim 10 wherein the antimetabolite is selected from antifolates, including methotrexate, tomudex and trimetrexate; 5-fluoropyrimidine derivatives, including 5-fluorouracil, floxuridine, ftorafur and capecitabine; and cytidine analogs, including cytarabine, azacitidine and gemcitabine.
14. Products according to claim 13 wherein the antimetabolite is selected from 5- fluorouracil or gemcitabine.
15. Products according to claim 10 comprising an acryloyl distamycin derivative of formula (I)
wherein:
Ri is a bromine or chlorine atom;
R2 is a group of formula (U)
wherein m is an integer from 0 to 2; n is an integer from 2 to 5; r is O or 1;
X and Y are, the same or different and independently for each heterocycUc ring, a nitrogen atom or a CH group;
G is phenylene, a 5 or 6 membered saturated or unsaturated heterocycUc ring with from 1 to 3 heteroatoms selected among N, O or S, or it is a group of formula (HI) below:
wherein Q is a nitrogen atom or a CH group and W is an oxygen or sulfur atom or it is a group NR3 wherein R3 is hydrogen or Cι-C alkyl; B is selected from the group consisting of
— CN . ~NR5R6 . - - CONR5R6 — NHCONR 5-F 6 wherein R is cyano, amino, hydroxy or Cι-C4 alkoxy; R5, R6 and R7, the same or different, are hydrogen or Cι-C4 alkyl.
16. Products according to claim 10 wherein the acryloyl distamycin derivative is selected from the group as defined in claim 9.
17. Use of an acryloyl distamycin derivative of formula (I), as defined in claim 1 or in any one of claims from 6 to 9 in the preparation of a medicament for use in combination therapy with an antimicrotubule agent and/or an antimetabolite in the treatment of tumors.
18. Use according to claim 17 wherein the medicament further comprises the said antimicrotubule agent and/or antimetabolite.
19. Use according to claim 17 or 18 wherein the acryloyl distamycin derivative is selected from the group as defined in claim 9.
20. Use according to any one of claims 17 to 19 wherein the tumor is selected from breast, ovary, lung, colon, kidney, stomach, pancreas, liver, melanoma, leukemia and brain tumors.
21. Use of an acryloyl distamycin derivative of formula (I), as defined in claim 1 or in any one of claims form 6 to 9 in the preparation of a medicament for use in combination therapy with an antimicrotubule agent and/or an antimetabolite in the
prevention or treatment of metastasis or in the treatment of tumors by inhibition of angiogenesis.
22. Use according to claim 21 wherein the medicament further comprises the said antimicrotubule agent and/or antimetabolite.
23. A method of treating a mammal, including humans, suffering from a neoplastic disease state, which method comprises administering to said mammal the acryloyl distamycin derivative of formula (I), as defined in claim 1 or any one of claims 6 to 9, an antimicrotubule agent and/or an antimetabolite, in amounts effective to produce a synergistic antineoplastic effect.
24. A method for lowering the side effects caused by antineoplastic therapy with an antineoplastic agent, in a mammal in need thereof including humans, the method comprising administering to said mammal a combined preparation comprising an antimicrotubule agent and/or an antimetabolite with an acryloyl distamycin derivative of formula (I), as defined in claim 1 or any one of claims from 6 to 7, in amounts effective to produce a synergistic antineoplastic effect.
25. A pharmaceutical composition according to claim 1 wherem the acryloyl distamycin derivative, optionally in the form of a pharmaceutically acceptable salt, is N-(5-{[(5-{[(5-{[(2-{[amino(imino)methyl]amino}ethyl)amino]carbonyl}-l-methyl- lH-pyrrol-3-yl)amino]carbonyl}-l-methyl-lH-pyrrol-3-yl)amino]carbonyl}-l-methyl- lH-pyrrol-3-yl)-4-[(2-bromoacryloyl)amino]-l-methyl-lH-pyrrole-2-carboxamide, and the antimetabolite is gemcitabine.
Applications Claiming Priority (3)
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| GB0015446.8 | 2000-06-23 | ||
| GBGB0015446.8A GB0015446D0 (en) | 2000-06-23 | 2000-06-23 | Combined therapy against tumors comprising substituted acryloyl distamycin derivates,taxanes and/or antimetabolites |
| PCT/EP2001/007060 WO2001097618A2 (en) | 2000-06-23 | 2001-06-20 | Combined therapy against tumors comprising substituted acryloyl distamycin derivatives, taxanes and/or antimetabolites |
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| MD36Z (en) * | 2008-12-02 | 2010-01-31 | Василе ЖОВМИР | Method for differential treatment of noninvasive ductal carcinoma in situ of mammary gland |
| MD35Z (en) * | 2008-12-02 | 2010-01-31 | Василе ЖОВМИР | Method for appreciating the risk of development of the noninvasive carcinoma in situ of the mammary gland |
| MD23Z (en) * | 2008-12-02 | 2010-01-31 | Василе ЖОВМИР | Method of differential treatment of noninvasive lobular mammary carcinoma in situ |
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| AT387013B (en) | 1985-07-16 | 1988-11-25 | Erba Farmitalia | METHOD FOR PRODUCING POLY-4-AMINOPYRROL -2-CARBOXAMIDO DERIVATIVES |
| GB8612218D0 (en) | 1986-05-20 | 1986-06-25 | Erba Farmitalia | Site specific alkylating agents |
| KR950013762B1 (en) | 1986-10-07 | 1995-11-15 | Boehringer Mannheim Italia | Pharmaceutical compositions having antineoplastic activity |
| GB8906709D0 (en) | 1989-03-23 | 1989-05-10 | Creighton Andrew M | Acryloyl substituted pyrrole derivatives |
| JP2919867B2 (en) | 1989-09-27 | 1999-07-19 | 千寿製薬株式会社 | Antitumor agent |
| CA2139638A1 (en) * | 1992-07-07 | 1994-01-20 | Nancy M. Gray | Methods of using (-) cisapride for the treatment of gastro-esophageal reflux disease and other disorders |
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-
2000
- 2000-06-23 GB GBGB0015446.8A patent/GB0015446D0/en not_active Ceased
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2001
- 2001-06-20 EE EEP200200688A patent/EE05359B1/en not_active IP Right Cessation
- 2001-06-20 CA CA002412054A patent/CA2412054A1/en not_active Abandoned
- 2001-06-20 DE DE60143681T patent/DE60143681D1/en not_active Expired - Lifetime
- 2001-06-20 ES ES01945297T patent/ES2317913T3/en not_active Expired - Lifetime
- 2001-06-20 SK SK1829-2002A patent/SK287398B6/en not_active IP Right Cessation
- 2001-06-20 NZ NZ523001A patent/NZ523001A/en unknown
- 2001-06-20 MX MXPA02012165A patent/MXPA02012165A/en active IP Right Grant
- 2001-06-20 AU AU6755301A patent/AU6755301A/en active Pending
- 2001-06-20 EP EP07122497A patent/EP1889624B1/en not_active Expired - Lifetime
- 2001-06-20 US US10/297,620 patent/US7642229B2/en not_active Expired - Fee Related
- 2001-06-20 JP JP2002503104A patent/JP2003535874A/en not_active Withdrawn
- 2001-06-20 CZ CZ20024107A patent/CZ20024107A3/en unknown
- 2001-06-20 BR BR0111814-5A patent/BR0111814A/en not_active Application Discontinuation
- 2001-06-20 PL PL363696A patent/PL200504B1/en not_active IP Right Cessation
- 2001-06-20 EP EP01945297A patent/EP1299110B1/en not_active Expired - Lifetime
- 2001-06-20 WO PCT/EP2001/007060 patent/WO2001097618A2/en not_active Ceased
- 2001-06-20 NZ NZ543318A patent/NZ543318A/en unknown
- 2001-06-20 CN CNB018116078A patent/CN100479824C/en not_active Expired - Fee Related
- 2001-06-20 IL IL15317801A patent/IL153178A0/en unknown
- 2001-06-20 AU AU2001267553A patent/AU2001267553B2/en not_active Ceased
- 2001-06-20 HK HK03106774.6A patent/HK1054506B/en not_active IP Right Cessation
- 2001-06-20 AT AT01945297T patent/ATE415165T1/en not_active IP Right Cessation
- 2001-06-20 EA EA200300059A patent/EA006709B1/en not_active IP Right Cessation
- 2001-06-20 AT AT07122497T patent/ATE491457T1/en not_active IP Right Cessation
- 2001-06-20 HU HU0301334A patent/HUP0301334A3/en unknown
- 2001-06-20 KR KR1020027017409A patent/KR100861668B1/en not_active Expired - Fee Related
- 2001-06-20 DE DE60136706T patent/DE60136706D1/en not_active Expired - Lifetime
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2002
- 2002-11-28 IL IL153178A patent/IL153178A/en not_active IP Right Cessation
- 2002-12-04 ZA ZA200209835A patent/ZA200209835B/en unknown
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